This might help explain the "Neuroimmune Puzzle"....
There is now evidence, the spike protein has been shown to occupy the nicotinic acetylcholine receptor. And they are expressed by both neuronal and nonneuronal cells throughout the body. These receptors help manage inflammation, autonomic functions like blood pressure and breathing, and cognition….
“Nicotinic receptors are involved in many processes that overlap with long COVID symptoms, such as memory, motor coordination, autonomic functions, and immune and vascular system regulation."
Take a guess where this quote comes from? Janna Moen, PhD, author of the paper you are discussing. She herself, suffered from Long COVID. She used nicotine patches and saw significant improvement in her dysautonomia and orthostatic intolerance. But is there any mention of the nicotinic acetylcholine receptors (nAChRs) in her study? Or that of nicotine showing promise in displacing the spike from these receptors? LOL!
Edit: On second look, buried within the study, there is a small mention of acetylcholine being released by T cells, which then act on other immune cells to inhibit inflammatory signaling. And that nicotine might help stimulate this pathway. You would think with her own personal experience, she would have elaborated a bit more on this. But instead, all she says is,.... " Although some case studies report a therapeutic effect of low-dose nicotine which may act by stimulating this pathway".
Using the 'find on page' tool, there is only one mention of nicotine in her entire study, and that was it.
I’m doing some work for publication related to brain inflammation if you want to chat about it.
This might help explain the "Neuroimmune Puzzle"... https://drphilipmcmillan.substack.com/p/long-covid-a-neuroimmune-puzzle-waiting/comment/128505278
Fascinating. Makes sense. What then?
This might help explain the "Neuroimmune Puzzle"....
There is now evidence, the spike protein has been shown to occupy the nicotinic acetylcholine receptor. And they are expressed by both neuronal and nonneuronal cells throughout the body. These receptors help manage inflammation, autonomic functions like blood pressure and breathing, and cognition….
“Nicotinic receptors are involved in many processes that overlap with long COVID symptoms, such as memory, motor coordination, autonomic functions, and immune and vascular system regulation."
Take a guess where this quote comes from? Janna Moen, PhD, author of the paper you are discussing. She herself, suffered from Long COVID. She used nicotine patches and saw significant improvement in her dysautonomia and orthostatic intolerance. But is there any mention of the nicotinic acetylcholine receptors (nAChRs) in her study? Or that of nicotine showing promise in displacing the spike from these receptors? LOL!
https://ohbaby.substack.com/p/nicotine-might-help-with-brain-fog
Edit: On second look, buried within the study, there is a small mention of acetylcholine being released by T cells, which then act on other immune cells to inhibit inflammatory signaling. And that nicotine might help stimulate this pathway. You would think with her own personal experience, she would have elaborated a bit more on this. But instead, all she says is,.... " Although some case studies report a therapeutic effect of low-dose nicotine which may act by stimulating this pathway".
Using the 'find on page' tool, there is only one mention of nicotine in her entire study, and that was it.
This is almost comical.
Because it's a result of toxicity from self assembling nanotechnolgy as I've clearly shown.
All these materials are toxic.